@string{ploscompbio = {PLoS Comput Biol}}
@string{nrn = {Nat Rev Neurosci}}
@string{arn = {Annu Rev Neurosci}}
@string{CurrBiol = {Curr Biol}}
@string{philtb 		= "Phil Trans R Soc Lond B"}
@string{prslb 		= "Proc R Soc Lond B"}
@string{pnas 		= "Proc Natl Acad Sci USA"}
@string{CurrOpNeuroBio = "Curr Opin Neurobiol"}
@string{NeuralComputation = "Neural Comput"}
@string{JNeuroscience 	= "J Neurosci"}
@string{JNeuroPhysiology = {J Neurophysiol}}
@string{vns = {Vis Neurosci}}

@article{beggs03,
  title =	 {Neuronal Avalanches in Neocortical Circuits},
  
author =	 {J. M. Beggs and D. Plenz},
  journal =	 JNeuroscience,
  year =	 2003,
  pages =	 {11167--11177},
  volume =	 23,
}

@article{berdondini2009,
  author =	 {Berdondini, L. and Imfeld, K. and Maccione, A. and
                  Tedesco, M. and Neukom, S. and Koudelka-Hep, M. and
                  Martinoia, S},
  year =	 2009,
  title =	 {Active pixel sensor array for high spatio-temporal
                  resolution electrophysiological recordings from
                  single cell to large scale neuronal networks},
  journal =	 {Lab Chip},
  volume =	 9,
  pages =	 {2644--2651},
}

@Article{blankenship2010,
  Author =	 "Blankenship, A. G.  and Feller, M. B. ",
  Title =	 "{Mechanisms underlying spontaneous patterned
                  activity in developing neural circuits}",
  Journal =	 nrn,
  Year =	 2010,
  Volume =	 11,
  Pages =	 "18--29",
  pmid =	 19953103,
  note =	 {\refbb Comprehensive review of the different
                  mechanisms generating spontaneous activity, not just
                  in retina, but other areas such as cochlea, spinal
                  cord and hippocampus.  It also highlights the robust
                  nature of spontaneous activity: when the circuitry
                  underlying activity generation is perturbed, other
                  circuits may compensate to restore activity
                  patterns.}
}

@Article{burgi94,
  title =	 {Model Based on Extracellular Potassium for
                  Spontaneous Synchronous Activity in Developing
                  Retinas},
  author =	 "P.-Y. Burgi and N. M. Grzywacz",
  journal =	 NeuralComputation,
  year =	 1994,
  volume =	 6,
  note =	 {\refb This was the first model of retinal wave
                  propagation.  Given the relatively low speed of wave
                  propagation, they suggested that fast gap junctions
                  were unlikely to be involved, but that slower
                  mechanisms, such as spread of extracellular
                  potassium, might be important.  Although subsequent
                  experiments have shown that different mechanisms are
                  involved, the authors were clear about which parts
                  of the model were underconstrained by biological
                  data at the time.},
  pages =	 "983--1004",
}

@Article{burgi94c,
  author =	 "P.-Y. Burgi and N. M. Grzywacz",
  title =	 "Model For The Pharmacological Basis Of Spontaneous
                  Synchronous Activity In Developing Retinas",
  journal =	 JNeuroscience,
  year =	 1994,
  volume =	 14,
  pages={7426--7439},
  abstract =	 "Spontaneous waves of bursts of action potentials
                  propagate across the ganglion-cell surface of
                  developing retinas. A recent biophysical model
                  postulated that this propagation is mediated by an
                  increase in extracellular K+, following its ejection
                  from ganglion cells during action
                  potentials. Moreover, the model hypothesized that
                  bursts might terminate due to the accumulation of
                  intracellular Ca2+ and the subsequent activation of
                  a Ca2+-dependent K+ conductance in the cells'
                  dendrites. Finally, the model proposed that an
                  excitatory synaptic drive causes a neuromodulation
                  of the waves' properties. To test the feasibility of
                  the model, we performed computer simulations of the
                  network of developing ganglion cells under control
                  and pharmacological-manipulation conditions. In
                  particular, we simulated the effects of neostigmine,
                  Cs+ and TEA, low Ca2+ concentrations, andCo2+. A
                  comparison of the simulations with
                  electrophysiological and pharmacological
                  experimental data recently obtained in turtles
                  (Sernagor and Grzywacz, 1993a), and cats and ferrets
                  (Meister et al.,1991; Wong et al., 1993), showed
                  that the model for the most part is consistent with
                  the behavior of developing retinas. Moreover,
                  modifications of the model to allow for GABAergic
                  inputs onto ganglion cells (Sernagor and Grzywacz,
                  1994) and poor [K+](out) buffering (Conners et al.,
                  1982) improved the model's fits. These results lent
                  further support to important roles of extracellular
                  K+ concentration and synaptic drive for the
                  propagation of waves.",
}

@Article{butts1999,
  author =	 {D. A. Butts and M. B. Feller and C. J. Shatz and
                  D. S. Rokhsar},
  title =	 {Retinal waves are governed by collective network
                  properties},
  journal =	 JNeuroscience,
  year =	 1999,
  volume =	 19,
  pages =	 {3580--3593}
}

@article{butts2007,
  title =	 {A Burst-Based ``{Hebbian}'' learning rule at
                  retinogeniculate synapses links retinal waves to
                  activity-dependent refinement},
  author =	 {D. A. Butts and P. O. Kanold and C. J. Shatz},
  journal =	 {PLoS Biol},
  volume =	 5,
  pages =	 {e61},
  year =	 2007
}

@article{butts2010,
  title =	 {The applicability of spike time dependent plasticity
                  to development},
  author =	 {Butts, D. A. and Kanold, P. O.},
  journal =	 {Front  Synaptic Neurosci},
  volume =	 2,
  year =	 2010,
  pages =	 30,
}

@Article{chalupa2009_nd,
  Author =	 "Chalupa, L. M. ",
  Title =	 {Retinal waves are unlikely to instruct the formation
                  of eye-specific retinogeniculate projections},
  Journal =	 "Neural Dev",
  Year =	 "2009",
  Volume =	 "4",
  Pages =	 "25",
  mynote =	 "\refb The exact nature of the role of spontaneous
                  activity patterns upon the development of neural
                  connectivity, such as eye-specific segregation, is
                  still widely debated.  Here the author argues
                  against an instructive/guidance role for activity in
                  the segregation of retinal inputs from different
                  eyes. See also companion article by Feller in the
                  same issue (cited below) for the opposite view.\textbf{TODO: expand}"
}

@article{conhaim10,
  author =	 {J. Conhaim and E. R. Cedarbaum and M. Barahimi and
                  J. G. Moore and M. I. Becker and H. Gleiss and
                  C. Kohl and W. J. Moody},
  title =	 {Bimodal septal and cortical triggering and complex
                  propagation patterns of spontaneous waves of
                  activity in the developing mouse cerebral cortex},
  journal =	 {Dev Neurobiol},
  volume =	 70,
  pages =	 {679--692},
  year =	 2010,
}

@Article{demas2006,
  author =	 {J Demas and B. T. Sagdullaev and E. Green and
                  L. Jaubert-Miazza and M. A. McCall and R. G. Gregg
                  and R. O. L. Wong},
  title =	 {Failure to maintain eye-specific segregation in \textit{nob},
                  a mutant with abnormally patterned retinal
                  activity.},
  journal =	 {Neuron},
  year =	 2006,
  volume =	 50,
  pages =	 {247--259}
}

@article{demas2011,
  title =	 {Vision drives correlated activity without patterned
                  spontaneous activity in developing \textit{Xenopus} retina},
  author =	 {Demas, J. A. and Payne, H. and Cline, H. T.},
  journal =	 {Dev Neurobiol},
  issn =	 {1932-846X},
  year =	 {in press},
  url =
                  {http://onlinelibrary.wiley.com/doi/10.1002/dneu.20880/abstract},
  note =	 {\refb Unlike recordings of spontaneous activity in
                  all other species recorded, correlated spontaneous
                  activity was not found in developing \emph{Xenopus} retina.
                  However, vision induces correlations similar to
                  spontaneous waves in other species, such as mice.}
}

@Article{eglen1999,
  author =	 {S. J. Eglen},
  title =	 {The role of retinal waves and synaptic normalization
                  in retinogeniculate development},
  journal =	 philtb,
  year =	 1999,
  volume =	 354,
  mynumber =	 1382,
  pages =	 {497--506}
}

@article{elliott1999,
  title =	 {A neurotrophic model of the development of the
                  retinogeniculocortical pathway induced by
                  spontaneous retinal waves},
  author =	 {Elliott, T. and Shadbolt, N. R.},
  journal =	 JNeuroscience,
  volume =	 19,
  pages =	 {7951--7970},
  year =	 1999,
  url =
                  {http://neuro.cjb.net/cgi/content/abstract/19/18/7951},
}

@article{famiglietti2010,
  title =	 {Development of excitatory and inhibitory
                  neurotransmitters in transitory cholinergic neurons,
                  starburst amacrine cells, and {GABAergic} amacrine
                  cells of rabbit retina, with implications for
                  previsual and visual development of retinal ganglion
                  cells},
  author =	 {Famiglietti, E. V. and Sundquist, S. J.},
  journal =	 vns,
  volume =	 27,
  pages =	 {19--42},
  issn =	 {1469-8714},
  year =	 2010,
}

@Article{feller1996,
  Author =	 "Feller, M. B.  and Wellis, D. P.  and Stellwagen, D.
                  and Werblin, F. S.  and Shatz, C. J. ",
  Title =	 "Requirement for cholinergic synaptic transmission in
                  the propagation of spontaneous retinal waves",
  Journal =	 "Science",
  Year =	 1996,
  Volume =	 272,
  Pages =	 "1182--1187",
}

@Article{feller2009_nd,
  Author =	 "Feller, M. B. ",
  Title =	 {Retinal waves are likely to instruct the formation
                  of eye-specific retinogeniculate projections},
  Journal =	 "Neural Dev",
  Year =	 "2009",
  Volume =	 "4",
  Pages =	 "24",
  mynote =	 {\refb See comments above for the Chalupa (2009)
                  reference.\textbf{TODO: expand}}
}

@article{feller97,
  title =	 {Dynamic processes shape spatiotemporal properties of
                  retinal waves},
  author =	 {Feller, M. B. and Butts, D. A. and Aaron, H. L. and
                  Rokhsar, D. S. and Shatz, C. J.},
  journal =	 {Neuron},
  year =	 1997,
  volume =	 19,
  sjenumber =	 2,
  pages =	 {293--306},
  note =	 {\refb This was the first theoretical model of
                  retinal waves developed along with novel
                  experimental data revealing the importance of
                  cholinergic transmission for retinal wave
                  propagation.  The model uses a two layer network of
                  SACs and RGCs (the readout layer) and can reproduce
                  interwave intervals, size and speed of waves
                  recorded with calcium imaging.  This work also first
                  proposed the spontaneous depolarization of SACs and
                  the refractory mechanism as being the key processes
                  for wave generation.}
}

@Article{ford2011,
  author =	 {K. J. Ford and A. L. F\'{e}lix and M. B. Feller},
  title =	 {Robust generation of retinal waves requires variable
                  participation of sparsely active interneurons},
  journal =	 {Nat Neurosci},
  year =	 {in review},
  note =         {\refbb This study combines theoretical predictions from the
  retinal model by Hennig et al. (2009) and novel experiments in the mouse retina
  during the period of cholinergic waves. Even though the study uses the same biophysical
  model, it makes one key novel prediction that variable participation of SACs is required in 
  subsequent waves. This prediction is then verified with two-photon calcium imaging. 
  Other differences with previous work in rabbit that the authors report is the less frequent spontaneous 
  depolarization of SACs and the ability of a single SAC to initiate a wave, thus suggesting a difference
  in the mechanisms regulating waves among different species. }
}

@article{gjorgjieva2009,
  title =	 {Burst-time-dependent plasticity robustly guides
                  {ON/OFF} segregation in the lateral geniculate
                  nucleus},
  author =	 {J. Gjorgjieva and T. Toyoizumi and S. J. Eglen},
  journal =	 ploscompbio,
  volume =	 5,
  pages =	 "e1000618",
  year =	 2009,
}

@article{godfrey2007,
  author =	 "Godfrey, K.  B. and Swindale, N. V.",
  title =	 {Retinal wave behavior through activity-dependent
                  refractory periods},
  journal =	 ploscompbio,
  year =	 2007,
  volume =	 3,
  pages =	 "e245",
  pmid =	 18052546,
  note =	 {\refb This work showed that a single-layer
                  consisting of a highly-connected SAC network is
                  sufficient to generate retinal wave behavior. The
                  model was evaluated against retinal wave data in
                  several species, including mouse, turtle, ferret,
                  rabbit and turtle. While the model relied on a SAC
                  network and cholinergic transmission, the model
                  formulation was relatively abstract allowing the
                  model to be adapted to retinal waves of different
                  nature (e.g. gap junction mediated waves or
                  glutamatergic waves) or even spontaneous activity
                  occurring in other parts of the brain.}
}

@Article{godfrey2009,
  Author =	 "Godfrey, K. B.  and Eglen, S. J.  and Swindale,
                  N. V. ",
  Title =	 "A multi-component model of the developing
                  retinocollicular pathway incorporating axonal and
                  synaptic growth",
  Journal =	 ploscompbio,
  Year =	 2009,
  Volume =	 5,
  Pages =	 "e1000600",
}

@Article{godfrey2009_review,
  author =	 {K. B. Godfrey and S. J. Eglen},
  title =	 {Theoretical models of spontaneous activity
                  generation and propagation in the developing retina},
  journal =	 {Mol Biosyst},
  year =	 2009,
  volume =	 5,
  pages =	 {1527--1535},
  note =	 {\refb The authors review in detail the Butts, Hennig
                  and Godfrey models with respect to the mechanisms
                  underlying retinal wave propagation, including the
                  role of neurotransmitters and second-messenger pathways.}
}

@article{hennig2009,
  title =	 {{Early-stage waves in the retinal network emerge
                  close to a critical state transition between local
                  and global functional connectivity}},
  author =	 {Hennig, M. H. and Adams, C. and Willshaw, D. and
                  Sernagor, E.},
  journal =	 JNeuroscience,
  volume =	 29,
  pages =	 {1077--1086},
  year =	 2009,
  note =	 {\refbb The most comprehensive of all retinal wave
                  models to date, consisting of biophysical neurons
                  with experimental parameters, synaptically-connected
                  in a network.  The model shows that retinal wave
                  networks operate in a critical regime between
                  complete synchrony and purely local activity,
                  regulated respectively by synaptic connectivity and
                  cell-intrinsic bursts. Wave size, wave duration and
                  interwave intervals follow power-law
                  distributions. The study provides a careful
                  quantitative comparison between model and
                  experimental data, and is the only one to use
                  experimental data from MEAs.}
}

@Article{huberman2008_arn,
  Author =	 "Huberman, A. D.  and Feller, M. B.  and Chapman, B.",
  Title =	 "{{M}echanisms underlying development of visual maps
                  and receptive fields}",
  Journal =	 arn,
  Year =	 2008,
  Volume =	 31,
  Pages =	 "479--509",
  note =	 {\refbb A thorough review which covers the main
                  principles behind the organization of the visual
                  system. The focus is on the formation of retinotopic
                  maps and receptive fields (orientation selectivity,
                  direction selectivity and ocular dominance columns).
                  The review discusses the two main mechanisms
                  believed to govern visual system development:
                  molecular guidance cues which set up the coarse map
                  for axons, and spontaneous activity which refines
                  exuberant connections and produces the adult refined
                  level of connectivity, further reinforced by
                  visually-evoked activity patterns. The authors also
                  compile a comprehensive list of observed map defects
                  in pharmacological and genetic perturbations in
                  mouse and ferret.},
}

@article{kerschensteiner2008,
  author =	 {D. Kerschensteiner and R. O. L. Wong},
  title =	 {A Precisely timed asynchronous pattern of {ON} and {OFF}
                  retinal ganglion cell activity during propagation of
                  retinal waves},
  journal =	 {Neuron},
  volume =	 58,
  year =	 2008,
  pages =	 {851--858},
}

@article{kumar10,
  Author =	 {A. Kumar and S. Rotter and A. Aertsen},
  Journal = {Nat Rev Neurosci},
  Pages =	 {615--627},
  Title =	 {Spiking activity propagation in neuronal networks:
                  reconciling different perspectives on neural coding},
  Volume ={11},
  Year ={2010},
 note={\refbb This review provides a unification of two key ideas which have been prominent in the theoretical study of networks: asynchronous (firing rate) propagation and synchronous (temporal code) propagation. The review describes the traditional feedforward network architecture used in previous studies and two modes of propagation which both have different advantages for information transmission and neural coding. Then it explores several network properties, such as synaptic connectivity and synaptic strength, and proposes that firing rate and synchronous modes of propagation are two extremes of a continuum, and that a network can support both modes, thus exploiting both as candidate neural codes. }
}

@Article{lee2002,
  author =	 {C. W. Lee and S. J. Eglen and R. O. L. Wong},
  title =	 {Segregation of on and off retinogeniculate
                  connectivity directed by patterned spontaneous
                  activity},
  journal =	 JNeuroPhysiology,
  year =	 2002,
  volume =	 88,
  pages =	 {2311-2321}
}

@article{lischalk09,
  author =	 {Lischalk, J. W. and Easton, C. R. and Moody, W. J.},
  year =	 2009,
  title =	 {Bilaterally propagating waves of spontaneous
                  activity arising from discrete pacemakers in the
                  neonatal mouse cerebral cortex},
  journal =	 {Dev Neurobiol},
  volume =	 69,
  pages =	 {407--414},
}

@Article{maffei90,
  author =	 "L. Maffei and L. Galli-Resta",
  title =	 "Correlation in the discharges of neighboring rat
                  retinal ganglion cells during prenatal life",
  journal =	 pnas,
  year =	 1990,
  volume =	 87,
  pages =	 "2861--2864",
}

@Article{odonovan1999,
  author =	 {M. J. {O'Donovan}},
  title =	 {The origin of spontaneous activity in developing
                  networks of the vertebrate nervous system},
  journal =	 CurrOpNeuroBio,
  year =	 1999,
  volume =	 9,
  pages =	 {94-104}
}

@article{rockhill09,
  author =	 {Rockhill, W. and Kirkman, J. L. and Bosma, M. M.},
  year =	 2009,
  title =	 {Spontaneous activity in the developing mouse
                  midbrain driven by an external pacemaker},
  journal =	 {Dev Neurobiol},
  volume =	 69,
  pages =	 {689--704},
}

@Article{sernagor1996,
  author =	 {E. Sernagor and N. M. Grzywacz},
  title =	 {Influence Of Spontaneous Activity And Visual
                  Experience On Developing Retinal Receptive Fields},
  journal =	 CurrBiol,
  year =	 1996,
  volume =	 6,
  pages =	 {1503-1508},
}

@InCollection{sernagor2006,
  author =	 {E. Sernagor},
  title =	 {Synaptogenesis and early neural activity.},
  booktitle =	 {Retinal Development},
  publisher =	 {Cambridge University Press},
  year =	 2006,
  pages =	 {265--287},
  editor =	 {E. Sernagor and S. J. Eglen and W. A. Harris and
                  R. O. L. Wong}
}

@InCollection{sernagor2011,
  author =	 {E. Sernagor and M. H. Hennig},
  title =	 {Retinal waves: underlying cellular mechanisms and
                  theoretical considerations},
  booktitle =	 {Comprehensive Developmental Neuroscience},
  publisher =	 {Elsevier},
  year =	 {in press},
  editor =	 {P. Rakic and J. Rubenstein}
}

@Article{stacy2005,
  author =	 {R. C. Stacy and J. Demas and R. W. Burgess and
                  J. R. Sanes and R. O. L. Wong},
  title =	 {Disruption and Recovery of Patterned Retinal
                  Activity in the Absence of Acetylcholine},
  journal =	 JNeuroscience,
  year =	 2005,
  volume =	 25,
  pages =	 {9347--9357}
}

@article{stafford2009,
  title =	 {Spatial-temporal patterns of retinal waves
                  underlying activity-dependent refinement of
                  retinofugal projections},
  author =	 {Stafford, B. K. and Sher, A. and Litke, A. M. and
                  Feldheim, D. A.},
  journal =	 {Neuron},
  volume =	 64,
  pages =	 {200--212},
  year =	 2009,
 }

@article{strata97,
  author =	 {Strata, F. and Atzori, M. and Molnar, M. and
                  Ugolini, G. and Tempia, F. and Cherubini, E.},
  year =	 1997,
  title =	 {{A pacemaker current in dye-coupled hilar
                  interneurons contributes to the generation of
                  giant GABAergic potentials in developing
                  hippocampus}},  
journal =	 {J Neurosci},
  volume =	 17,
  pages =	 {1435--1446},
}

@Article{syed2004,
  Author =	 {Syed, M. M.  and Lee, S.  and Zheng, J.  and Zhou,  Z. J. },
  Title =	 {Stage-dependent dynamics and modulation of
                  spontaneous waves in the developing rabbit retina},
  Journal =	 "J Physiol",
  Year =	 2004,
  Volume =	 560,
  Pages =	 {533--549},
}

@article{tabak00,
  author =	 {J. Tabak and W. Senn and M. J. O'Donovan and
                  J. Rinzel},
  title =	 {Modeling of spontaneous activity in the developing
                  spinal cord using activity-dependent depression in
                  an excitatory network},
  journal =	 {J Neurosci},
  year =	 2000,
  volume =	 20,
  pages =	 {3041--3056},
}

@article{tabak10,
  title =	 {Mechanism for the universal pattern of activity in
                  developing neuronal networks},
  author =	 {J. Tabak and M. Mascagni and R. Bertram},
  journal =	 {J Neurophysiol},
  year =	 2010,
  pages =	 {2208--2221},
  volume =	 103,
}

@article{thivierge2009_nn,
  title =	 {How does non-random spontaneous activity contribute
                  to brain development?},
  author =	 {Thivierge, J. P.},
  journal =	 {Neural Networks},
  volume =	 22,
  pages =	 {901--912},
  year =	 2009,
}

@Article{willshaw1976,
  author =	 "D. J. Willshaw and C. {von der Malsburg}",
  title =	 "How patterned neural connections can be set up by
                  self-organization",
  journal =	 prslb,
  year =	 1976,
  volume =	 194,
  pages =	 "431--445",
}


@article{wong99,
  author =	 {R. O. L. Wong},
  title =	 {Retinal Waves and Visual System Development},
  journal =	 arn,
  volume =	 22,
  pages =	 {29--47},
  year =	 1999,
}

@article{zheng2006_ahp,
  title =	 {A transient network of intrinsically bursting
                  starburst cells underlies the generation of retinal
                  waves},
  author =	 {Zheng, J. and Lee, S. and Zhou, Z. J.},
  journal =	 {Nat Neurosci},
  volume =	 {9},
  pages =	 {363--371},
  year =	 {2006},
  note =	 {\refb The first experimental study to show that
                  rabbit SACs have long-lasting (on the order of
                  seconds) after-hyperpolarizations, which regulate
                  the refractory period of SACs, as opposed to a
                  depletion of neurotransmitter as thought
                  previously. The authors also estimated the number of
                  neighboring SACs from which each SAC receives
                  synaptic input during a wave, thus suggesting that
                  many SACs must fire together to generate a wave. The
                  newer retinal waves models (Godfrey and Hennig) have
                  based their assumptions on the findings from this
                  study.}
}



